摘要: 并发症的发生主要与DHCA脑缺血及再灌注损伤有关，其中谷氨酸的兴奋性神经毒性为造成损伤的重要原因，谷氨酸作用于N-甲基-D-天冬氨酸(NMDA)受体，导致大量Ca2+内流，激活NO合酶，NO的合成增加，因而NO在神经系统缺血再灌注损伤的作用逐渐受到重视。一、NO的特性1。 NO的生化和生理特性NO在体内的生理作用非常广泛，对血液......
作者单位： 100029 北京，首都医科大学附属安贞医院体外循环科
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